Search results for "γ secretase"

showing 6 items of 6 documents

SAR-studies of γ-secretase modulators with PPARγ-agonistic and 5-lipoxygenase-inhibitory activity for Alzheimer’s disease

2014

Abstract We present the design, synthesis and biological evaluation of compounds containing a 2-(benzylidene)hexanoic acid scaffold as multi-target directed γ-secretase-modulators. Broad structural variations were undertaken to elucidate the structure–activity-relationships at the 5-position of the aromatic core. Compound 13 showed the most potent activity profile with IC50 values of 0.79 μM (Aβ42), 0.3 μM (5-lipoxygenase) and an EC50 value of 4.64 μM for PPARγ-activation. This derivative is the first compound exhibiting low micromolar to nanomolar activities for these three targets. Combining γ-secretase-modulation, PPARγ-agonism and inhibition of 5-lipoxygenase in one compound could be a …

Clinical BiochemistryPharmaceutical SciencePeroxisome proliferator-activated receptorInflammationDiseasePharmacologyInhibitory postsynaptic potentialBiochemistryStructure-Activity Relationshipchemistry.chemical_compoundAlzheimer DiseaseDrug DiscoverymedicineHumansLipoxygenase Inhibitorsγ secretaseCaproatesMolecular BiologyHexanoic acidchemistry.chemical_classificationArachidonate 5-LipoxygenasebiologyOrganic ChemistryPPAR gammachemistryBiochemistryArachidonate 5-lipoxygenasebiology.proteinMolecular MedicineAmyloid Precursor Protein Secretasesmedicine.symptomDerivative (chemistry)Bioorganic & Medicinal Chemistry Letters
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Gamma-secretase modulation with Abeta42-lowering nonsteroidal anti-inflammatory drugs and derived compounds.

2006

The amyloid-beta (Abeta) peptides and specifically the highly amyloidogenic isoform Abeta42 appear to be key agents in the pathogenesis of familial and sporadic forms of Alzheimer's disease (AD). The final step in the generation of Abeta from the amyloid precursor protein is catalyzed by the multiprotein complex gamma-secretase, which constitutes a prime drug target for prevention and therapy of the disease. However, highly potent gamma-secretase inhibitors that block formation of all Abeta peptides have provoked troubling side effects in preclinical animal models of AD. This toxicity can be readily explained by the promiscuous substrate specificity of gamma-secretase and its essential role…

Gene isoformendocrine systemClinical Trials as TopicNonsteroidalAmyloid beta-Peptidesmedicine.drug_classAnti-Inflammatory Agents Non-SteroidalPharmacologyIbuprofenAmyloid β peptideAnti-inflammatoryPathogenesischemistry.chemical_compoundNeurologychemistryAlzheimer DiseasemedicineAnimalsHumansNeurology (clinical)γ secretaseAmyloid Precursor Protein SecretasesGamma secretasemedicine.drugNeuro-degenerative diseases
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Loss of γ-secretase function impairs endocytosis of lipoprotein particles and membrane cholesterol homeostasis

2008

Membrane cholesterolChemistryOrganic ChemistryCell Biologyγ secretaseEndocytosisMolecular BiologyBiochemistryLRP1HomeostasisFunction (biology)LipoproteinCell biologyChemistry and Physics of Lipids
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P3‐344: γ‐secretase‐dependent APP processing occurs either at the plasma membrane or in the endocytic compartments independent of the APP wild‐type o…

2008

Psychiatry and Mental healthCellular and Molecular NeuroscienceMembraneDevelopmental NeuroscienceEpidemiologyChemistryHealth PolicyEndocytic cycleWild typeNeurology (clinical)γ secretaseGeriatrics and GerontologyCell biologyAlzheimer's & Dementia
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Discovery of γ-secretase modulators with a novel activity profile by text-based virtual screening.

2012

We present an integrated approach to identify and optimize a novel class of γ-secretase modulators (GSMs) with a unique pharmacological profile. Our strategy included (i) virtual screening through application of a recently developed protocol (PhAST), (ii) synthetic chemistry to discover structure–activity relationships, and (iii) detailed in vitro pharmacological characterization. GSMs are promising agents for treatment or prevention of Alzheimer’s disease. They modulate the γ-secretase product spectrum (i.e., amyloid-β (Aβ) peptides of different length) and induce a shift from toxic Aβ42 to shorter Aβ species such as Aβ38 with no or minimal effect on the overall rate of γ-secretase cleavag…

PyridinesPyridonesMolecular Sequence DataPeptideComputational biologyCHO CellsBiochemistryStructure-Activity RelationshipAlzheimer DiseaseCricetinaeAnimalsHumansγ secretaseAmino Acid Sequencechemistry.chemical_classificationVirtual screeningActivity profileAmyloid beta-PeptidesChemistryGeneral MedicineIntegrated approachIn vitroMinimal effectDrug DesignMolecular MedicineAmyloid Precursor Protein SecretasesACS chemical biology
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P4‐192: Mechanism of γ‐secretase cleavage: Evidence for independent generation of Aβ42 and Aβ38 peptide species

2008

chemistry.chemical_classificationCleavage factorEpidemiologyChemistryStereochemistryHealth PolicyPeptideCleavage (embryo)Psychiatry and Mental healthCellular and Molecular NeuroscienceDevelopmental NeuroscienceNeurology (clinical)γ secretaseGeriatrics and GerontologyAlzheimer's & Dementia
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